Expression of LOX in Colorectal Cancer Tissues and Its Relationship with Progress and Prognosis
Abstract
To analyze the expression of lysyl oxidase (LOX) in colorectal cancer and its relationship with clinicopathological characteristics, prognosis, and its progress. Methods 82 cases of colorectal tumor paraffin-embedded specimens and paired tumor-adjacent tissues were collected, and data of clinicopathological characteristics and prognosis of these patients were also recorded from 2009.1 to 2010.5. Expressions of LOX, hypoxia inducible factor-1α (HIF-1α), matrix metalloproteinase (MMP)-2, MMP-7 were determined by immunohistochemistry. Then relationship between LOX and clinicopathological characteristics and prognosis was explored, and relationship between LOX and HIF-1α, MMP-2, MMP-7 were investigated. Results Expressions of LOX was stronger in tumors than in tumor-adjacent tissues (P<0.05). Cancer tissues with overexpressed LOX had later clinical stages, deeper tumor invasion, and more metastatic lymph nodes (all P<0.05). The result also showed that patients with overexpression of LOX had poorer prognosis, and overexpression of LOX was independent factor for prognosis in COX survival analysis. Expression of HIF-1α, MMP-2, MMP-7 in colorectal cancer was stronger than in tumor-adjacent tissues (P<0.05). Positive relationship was found between LOX and HIF-1α, MMP-2, MMP-7 proteins (P<0.05). Conclusion LOX was overexpressed in colorectal cancer tissues, and was associated with the progression of colorectal cancer. LOX may be involved in the progress of colorectal cancer by regulating HIF-1α, MMP-2, MMP-7 protein expression.
Keywords: Colorectal cancer, Lysyl oxidase (LOX) gene, Clinicopathological characteristics, Prognosis, Invasion, metastasis
Full Text:
PDFReferences
MILLER BW, MORTON JP. PINESE M. et al. Targeting the LOX/hypoxia axis reverses many of the features that make pancreatic cancer deadly: inhibition of LOX abrogates metastasis and enhances drug efficacy. EMBO Mol Med,2015. 7(8):1063-1076.
LIU J, PING W, ZU Y, et al. Correlations of lysyl oxidase with MMP2/MMP9 expression and its prognostic value in non¬small cell lung cancer. Int J Clin Exp Pathol ,2014 ,7( 9) ;6040- 6047.
KIM HY.CHO HK, YOO SK, et al. Hepatic STAMP2 deceases hepatitis В virus X protein-associated metabolic deregulation. Exp Mol Med.2012,44(10); 622-632.
KIM KH. SHIN HJ, KIM K, et al. Hepatitis В virus X protein induces hepatic steatosis via transcriptional activation of SREBP1 and PPARgamma. Gastroenterolory, 2007, 132 (5);1955-1967.
LAI H. JIN Q, LIN Y, et al. Combined use of lysyl oxidase, carcino-embryonic antigen, and carbohydrate antigens improves the sensitivity of biomarkers in predicting lymph node metastasis and peritoneal metastasis in gastric cancer. Tumour Biol,2014 ,35( 10) : 10547-10554.
GAO X, ZHANG S, ZHU Z. Lysyl oxidase rsl800449 polymorphism and cancer risk among Asians: evidence from a meta-analysis and a case-control study of colorectal cancer. Mol Genet Genomics,2015 »290( 1) :23-28.
PERRYMAN L. ERLER JT. Lysyl oxidase in cancer research. Future Oncol,2014 ,10(9): 1709-1717. PAPADANTONAKIS N, MATSUURA S, RAVID K. Megakaryocyte pathology and bone marrow fibrosis; the lysyl oxidase connection. Blood.2012,120(9); 1774-1781.
JI F, WANG Y, QIU L, et al. Hypoxia inducible factor la- mediated LOX expression correlates with migration and invasion in epithelial ovarian cancer. Int J Oncol, 2013, 42 (5):1578-1588.
AGRA N, CIDRE F, GARCIA-GARCIA L, et al. Lysyl oxidase is downregulated by the EWS/FLI1 oncoprotein and its propeptide domain displays tumor supressor activities in Ewing sarcoma cells. PLoS One, 2013, 8 ( 6): e66281 [2016- 10-07]. http;//doi. org/10. 1371/journal, pone. 0066281.
WANG Y, MA J, SHEN H, et al. Reactive oxygen species promote ovarian cancer progression via the HIF-la/LOX/E- cadherin pathway. Oncol Rep,2014 ,32(5);2150-2158.
LIU JL, WEI W. TANG W. et al. Silencing of lysyl oxidase gene expression by RNA interference suppresses metastasis of breast cancer. Asian Рас J Cancer Prev,2012,13(7);3507- 3511.
Refbacks
- There are currently no refbacks.



